Study Reveals Mechanism Behind Failed Remyelination in MS

Updated December 18, 2018

Research by Fraser J. Sim, PhD, associate professor of pharmacology and toxicology, suggests that the spontaneous regeneration of myelin, the brain’s fatty insulator that keeps neurons communicating, could lead to a novel approach to developing treatments for multiple sclerosis and other inflammatory diseases. "The idea that pathological quiescence of progenitors could prevent regeneration in MS is distinct from the current pre-clinical strategies making their way into trial," he said.